Tetracosactide acetate
CAS No. 60189-34-6
Tetracosactide acetate( Tetracosactrin acetate )
Catalog No. M28050 CAS No. 60189-34-6
Cosyntropin (acetate) stimulates the release of corticosteroids such as cortisol from the adrenal gland.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
Size | Price / USD | Stock | Quantity |
5MG | 177 | Get Quote |
|
10MG | 290 | Get Quote |
|
25MG | 492 | Get Quote |
|
50MG | 709 | Get Quote |
|
100MG | 972 | Get Quote |
|
200MG | Get Quote | Get Quote |
|
500MG | Get Quote | Get Quote |
|
1G | Get Quote | Get Quote |
|
Biological Information
-
Product NameTetracosactide acetate
-
NoteResearch use only, not for human use.
-
Brief DescriptionCosyntropin (acetate) stimulates the release of corticosteroids such as cortisol from the adrenal gland.
-
DescriptionCosyntropin (acetate) stimulates the release of corticosteroids such as cortisol from the adrenal gland.
-
In Vitro——
-
In Vivo——
-
SynonymsTetracosactrin acetate
-
PathwayOthers
-
TargetOther Targets
-
Recptor——
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number60189-34-6
-
Formula Weight2993.5
-
Molecular FormulaC136H210N40O31S.XC2H4O2
-
Purity>98% (HPLC)
-
Solubility——
-
SMILESN/A
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
1.Zhang M, et al. Improving the flame retardancy of poly(lactic acid) using an efficient ternary hybrid flame retardant by dual modification of graphene oxide with phenylphosphinic acid and nano MOFs. J Hazard Mater. 2020 Feb 15;384:121260.
molnova catalog
related products
-
Glyasperin F
Glyasperin F has antinociceptive, and anti-inflammatory effects, it could inhibit NO production in RAW 264.7 macrophages to some extent.
-
Traumatic Acid
Traumatic Acid is a product of the hydroperoxide lyase pathway in plants. Potential as a wound healing agent that stimulates cell division near a wound site to form a protective callus.
-
kaempferide
Kaempferide triglycoside inhibits the proliferation of native and estrogen receptor beta overexpressing colon cancer cells through a mechanism not mediated by ligand binding dependent estrogen receptor activation.